Anatomy and Dynamics
In order to explain the Anatomy and Dynamics of the Integral System the following page has been separated into the following sections:
Function and Dysfunction
The Integral Theory describes normal bladder function as having two stable states: closed (continence) and opening (micturition). These states are deemed to be stable because they are the resultants of a balance of forces.
Three muscle forces interact during closure (fig 2-13) and two muscle forces interact during opening (fig 2-14). If the efficacy or balance of these muscle forces is disrupted through damage to their ligamentous anchoring points, dysfunction may result in both closure (incontinence) or opening (abnormal emptying).
During effort (fig 2-13) the distal vagina is stretched forwards by PCM. The upper vagina and bladder base are stretched down and back by LP and LMA. PCM and LP contract against the pubourethral ligament (PUL). LMA contracts against the uterosacral ligaments (USL). The broken lines represent the resting position of the bladder.
During micturition (fig. 2-14), PCM relaxes.The stretch receptors activate the micturition reflex. The whole system is stretched down and back by LP and LMA, opening out the outflow tract. The detrusor contracts to expel urine. The broken lines represent the closed position of the bladder.
Note that, although the terms ‘opening’ and ‘micturition’ are used interchangeably, they are not exactly the same. ‘Micturition’ is driven by a neurological reflex whereas the ‘open’ state of the urethra may result from entirely mechanical factors, for example, ‘genuine stress iincontinence’.
Structure and Form
Structure and form in the pelvic floor arise from the interaction of muscles, nerves and ligaments acting on the pelvic organs. The vagina and ligaments must be stretched to their limit of extension to attain the strength required to carry loads. Unequal balance of the forces may stretch the system one way or the other, thereby affecting opening or closure. The ‘suspension bridge’ analogy (figs 1-05 and 1-08) is used to illustrate how structure and form (and thereby function) arise from a system in balanced tension.
Delineating Zones of Damage
Causes of Dysfunction
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The circles represent the foetal head descending through the birth canal. Damage to one or more ligaments leads to imbalance of the system and hence dysfunction. Congenitally defective collagen may also cause dysfunction.
The middle zone extends from the bladder neck to the cervix or hysterectomy scar. It contains two structures which may be damaged: the pubocervical fascia (PCF),cardinal ligament/anterior cervical ring and the arcus tendineus fasciae pelvis (ATFP).
The posterior zone extends from the cervix or hysterectomy scar to the perineal body. The uterosacral ligaments (USL), rectovaginal fascia (RVF) and perineal body (PB) are the key posterior zone structures which may be damaged.
Locating the damaged structures within the zones
Within each zone are key structures which facilitate normal pelvic floor function. If any of these is damaged, dysfunction may result. The Integral Theory delineates nine key structures in which damage may occur. The three zones each contain three main stuctures which may be damaged (fig 1-10).
They are:
Anterior zone
Excess Laxity
1. External urethral ligament (EUL)
2. Suburethral vagina (hammock)
3. Pubourethral ligament (PUL)
Middle zone
Excess Laxity
4. Arcus tendineus fascia pelvis (ATFP)
5. Pubocervical fascial defect (PCF) (cystocoele)
6. Cardinal ligament/cervical ring attachment of PCF (high cystocoele) ‘transverse defect’
Excessive tightness
‘ZCE’ - Excessive tightness in the ‘Zone of critical elasticity’ (ZCE) (Iatrogenic
cause: excess scarring; excess elevation with colposuspension)
Posterior zone
Excess Laxity
7. Uterosacral ligaments (USL)
8. Rectovaginal fascial (RVF)
9. Perineal body (PB).
Note that the ZCE is part of the PCF. It is included here as a separate structure to emphasize the importance of maintaining elasticity at the bladder neck area of the vagina (ZCE) during surgery. It is in the ZCE where the ‘tethered vagina syndrome’ originates. The ‘tethered vagina syndrome’ is described fully later in this book. Briefly, it is caused by loss of elasticity at ZCE from vaginal or bladder neck elevation surgery due to scarring or excessive bladder neck elevation. It is entirely iatrogenic and can be included under defect 5 (pubocervical fascia), except that it specifies excessive tightness, not laxity.